Shock Index Calculator
Shock Index = HR ÷ SBP. Enter DBP for Modified Shock Index (HR ÷ MAP). Normal SI <0.7.
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The Shock Index (SI) is a simple bedside haemodynamic ratio calculated by dividing the heart rate by the systolic blood pressure. First described by Allgöwer and Burri in 1967, it was originally developed to assess blood loss in trauma patients. A rising Shock Index reflects the physiological compensation that occurs when perfusion pressure falls: the body increases heart rate (tachycardia) to maintain cardiac output as systolic blood pressure drops, creating a ratio that rises progressively with haemodynamic compromise. In a healthy resting adult, the SI is typically between 0.5 and 0.7 — heart rate is lower than or similar to systolic blood pressure. As circulating volume falls or vasomotor tone is lost, heart rate climbs while systolic pressure drops, pushing the SI above 1.0 in moderate-to-severe shock states. The Shock Index is used across a wide range of acute clinical scenarios. In trauma, an SI greater than 1.0 at triage predicts the need for massive transfusion, early activation of major haemorrhage protocols, and increased 30-day mortality. In obstetric haemorrhage, the SI may detect significant blood loss earlier than conventional vital signs because pregnant women compensate so effectively that standard thresholds can be falsely reassuring. In upper gastrointestinal bleeding, the SI at presentation predicts rebleeding and in-hospital mortality. In sepsis and pulmonary embolism, an elevated SI identifies patients at high risk of decompensation. The Modified Shock Index (MSI), which substitutes mean arterial pressure (MAP) for systolic blood pressure, offers greater sensitivity for detecting haemodynamic instability in some populations, particularly in trauma and post-partum haemorrhage.
Shock Index (SI) = Heart Rate (bpm) / Systolic Blood Pressure (mmHg). Modified Shock Index (MSI) = Heart Rate (bpm) / Mean Arterial Pressure (mmHg), where MAP = (SBP + 2 × DBP) / 3. Normal SI: 0.5–0.7. Elevated: >0.8. Abnormal: >1.0. Critical: >1.4.
- 1Measure the patient's heart rate (HR) in beats per minute and systolic blood pressure (SBP) in mmHg using non-invasive monitoring.
- 2Divide HR by SBP: SI = HR / SBP. No correction factors, age adjustments, or laboratory values are needed.
- 3Interpret: SI 0.5–0.7 is normal for a resting adult. SI 0.8–1.0 suggests mild haemodynamic compromise. SI >1.0 indicates significant shock and should trigger urgent assessment and intervention.
- 4In trauma: SI >1.0 at triage activates major haemorrhage protocol consideration; SI >1.4 is associated with haemorrhagic shock requiring immediate transfusion.
- 5For obstetric haemorrhage: use the SI ≥1.0 threshold earlier and more aggressively, as pregnant and post-partum patients compensate until very late-stage blood loss.
- 6Calculate the Modified Shock Index if MAP is available: MSI = HR / MAP. An MSI >1.4 has higher sensitivity for predicting massive transfusion than SI alone in some studies.
- 7Re-measure the SI after each intervention (fluid bolus, blood transfusion, vasopressor initiation) to track haemodynamic response — a falling SI indicates improvement; a persistent or rising SI signals ongoing compromise.
Well within the normal range of 0.5–0.7.
This is a healthy resting vital sign pattern. The SI confirms adequate haemodynamic reserve with no evidence of shock physiology. No intervention is required.
SI approaching 1.0 — early haemorrhage warning in trauma context.
Although the blood pressure appears 'acceptable', the elevated heart rate pushes the SI close to 1.0. In a trauma patient, this may indicate Class II haemorrhage (750–1500 mL blood loss) and warrants close monitoring, IV access, and preparation for transfusion.
SI >1.4 in obstetric haemorrhage — activate major haemorrhage protocol immediately.
An SI of 1.42 in a post-partum patient indicates substantial blood loss with failing compensation. The obstetric Shock Index threshold (≥1.0) is lower than in general trauma. Immediate actions: call the major haemorrhage team, activate massive transfusion protocol, administer tranexamic acid, prepare for surgical/interventional haemostasis.
Both SI >1.0 and MSI >1.4 confirm haemodynamic compromise despite a borderline systolic BP.
A systolic blood pressure of 95 mmHg might be considered 'borderline' by some practitioners, but both the SI and MSI are markedly elevated, confirming inadequate perfusion. The MAP of 68 mmHg is at the threshold for organ hypoperfusion. This patient requires urgent vasopressor support and source control.
Trauma triage in emergency departments and pre-hospital settings to identify patients requiring massive transfusion protocol activation., where accurate shock index analysis through the Shock Index supports evidence-based decision-making and quantitative rigor in professional workflows
Monitoring haemodynamic response to resuscitation in real time — a falling Shock Index after fluid or blood transfusion confirms improvement., where accurate shock index analysis through the Shock Index supports evidence-based decision-making and quantitative rigor in professional workflows
Detecting significant post-partum haemorrhage earlier than conventional vital sign thresholds in obstetric emergencies., where accurate shock index analysis through the Shock Index supports evidence-based decision-making and quantitative rigor in professional workflows
Risk stratification in acute upper gastrointestinal bleeding to identify patients requiring urgent endoscopy., where accurate shock index analysis through the Shock Index supports evidence-based decision-making and quantitative rigor in professional workflows
Rapid bedside identification of haemodynamic instability in sepsis, pulmonary embolism, and ruptured aortic aneurysm when more complex scoring is impractical.
Obstetric haemorrhage — lower threshold applies
Pregnant and post-partum women have a physiologically elevated baseline blood volume and heart rate, and they compensate for haemorrhage more effectively than non-pregnant women. This means conventional vital sign thresholds significantly underestimate blood loss. An SI ≥0.9 in the post-partum period should be treated with the same urgency as SI >1.0 in a non-pregnant adult. RCOG recommends using the SI as a primary triage tool in all cases of suspected post-partum haemorrhage.
Beta-blocker and rate-limiting medication use
Patients taking beta-blockers, rate-limiting calcium channel blockers, or digoxin may not produce the expected tachycardia in response to haemorrhage or sepsis. In such patients, the SI will be artificially low despite significant physiological compromise. Clinicians must maintain a high index of suspicion and use additional markers — lactate, base excess, urine output — to assess perfusion adequacy.
Neurogenic shock
In high spinal cord injury (cervical or upper thoracic), loss of sympathetic tone causes both bradycardia and hypotension, producing an SI that may be <1.0 despite severe haemodynamic compromise. This is paradoxical relative to haemorrhagic shock and reflects a different pathophysiology. In this context, the SI must be interpreted alongside the clinical scenario and mechanism of injury.
Upper GI bleeding
In acute upper gastrointestinal bleeding, an SI >1.0 at presentation is associated with significantly increased risk of rebleeding, need for endoscopic intervention, blood transfusion requirement, and in-hospital mortality. Some guidelines recommend using SI >1.0 as an indication for urgent (same-night) endoscopy rather than waiting until the next morning.
Pulmonary embolism
In acute pulmonary embolism, the Shock Index correlates with right ventricular strain and haemodynamic compromise. An SI >1.0 in suspected PE is associated with higher 30-day mortality and may support the use of systemic thrombolysis or catheter-directed therapy rather than anticoagulation alone, particularly when combined with echocardiographic evidence of RV dysfunction.
| SI Value | Category | Estimated Blood Loss | Clinical Action |
|---|---|---|---|
| 0.5–0.7 | Normal | Minimal | Routine monitoring; no immediate intervention required |
| 0.8–1.0 | Mild shock | Up to ~15–30% (Class I–II) | Close monitoring, IV access, consider fluid challenge, investigate cause |
| 1.0–1.4 | Moderate shock | ~30–40% (Class III) | Urgent resuscitation, blood products, activate transfusion protocol |
| >1.4 | Severe/critical shock | >40% (Class IV) | Immediate massive transfusion protocol, surgical/interventional haemostasis, ITU |
What is the Shock Index?
The Shock Index is the ratio of heart rate divided by systolic blood pressure. It was developed in 1967 as a simple bedside tool to detect haemodynamic instability before conventional blood pressure thresholds are breached. A normal SI is 0.5–0.7; values above 1.0 indicate significant shock and prompt urgent clinical intervention.
Why is the Shock Index useful when blood pressure seems normal?
Systolic blood pressure can remain within normal range through compensatory tachycardia even as blood volume falls by up to 30% (Class II haemorrhage). The Shock Index detects this compensation by incorporating heart rate, which rises much earlier than blood pressure falls. An SI of 0.9–1.0 with a 'normal' blood pressure may still signal significant occult haemorrhage.
What is the Modified Shock Index (MSI) and when is it preferred?
The Modified Shock Index uses mean arterial pressure (MAP) instead of systolic blood pressure: MSI = HR / MAP. MAP reflects average perfusion pressure throughout the cardiac cycle and is a better predictor of end-organ perfusion. MSI >1.4 has shown higher sensitivity than SI >1.0 for predicting massive transfusion need in some trauma and obstetric studies.
What Shock Index value should trigger a major haemorrhage protocol?
In trauma, an SI >1.0 at triage is commonly used to activate massive transfusion protocol consideration, with SI >1.4 indicating high likelihood of need for blood products. In obstetric haemorrhage, a lower threshold of SI ≥1.0 is recommended by RCOG and other bodies. Institutional protocols vary; always follow local guidelines.
Can the Shock Index be falsely normal in serious illness?
Yes. Patients on beta-blockers or rate-limiting calcium channel blockers may not mount an appropriate tachycardia, causing the SI to underestimate haemodynamic compromise. Similarly, a patient with pre-existing hypertension may still have an SI <1.0 even with significant blood loss because their 'normal' SBP is much higher than average. This is particularly important in the context of shock index calculations, where accuracy directly impacts decision-making. Professionals across multiple industries rely on precise shock index computations to validate assumptions, optimize processes, and ensure compliance with applicable standards. Understanding the underlying methodology helps users interpret results correctly and identify when additional analysis may be warranted.
Is the Shock Index validated in paediatric patients?
The standard SI thresholds apply to adults. Paediatric normal vital signs differ significantly by age — children have higher heart rates and lower blood pressures at baseline — so the adult SI thresholds should not be applied to children without age-appropriate adjustment. Paediatric-specific versions have been studied but are not yet universally standardised.
How does the Shock Index compare to other triage tools like NEWS2?
The Shock Index is faster to calculate (two values only) and particularly sensitive for haemorrhagic shock. NEWS2 (National Early Warning Score 2) incorporates six physiological parameters and is better validated across a broader range of acutely ill patients including sepsis, respiratory failure, and neurological deterioration. In trauma and haemorrhage, SI is complementary to — and often faster than — composite scores.
Can the Shock Index predict mortality?
Yes. In multiple prospective studies, an SI >1.0 at presentation is independently associated with increased 30-day in-hospital mortality in trauma, gastrointestinal bleeding, and obstetric haemorrhage. The Shock Index is a continuous variable — higher values carry progressively worse prognosis. It has been incorporated into composite scores like the OASIS and ABC (Assessment of Blood Consumption) trauma scores.
Ammattilaisen vinkki
Use the Shock Index as a dynamic serial tool rather than a single measurement. Plot HR and SBP against time during resuscitation — a falling SI after transfusion or fluids confirms haemodynamic response. A persistent SI >1.0 despite initial resuscitation should prompt urgent investigation for ongoing haemorrhage, tension pneumothorax, or refractory shock requiring surgical or advanced intervention.
Tiesitkö?
The Shock Index was first proposed by Martin Allgöwer and Carlo Burri in a 1967 paper examining World War II-era field trauma data. It was designed to be calculated without any equipment — just a watch and a finger on the radial pulse. Over 55 years later, it remains one of the most commonly used and validated rapid triage tools in emergency medicine, and now forms part of the Assessment of Blood Consumption (ABC) trauma score used to activate massive transfusion protocols worldwide.
Viitteet
- ›Allgöwer M, Burri C — Shock Index (Dtsch Med Wochenschr, 1967)
- ›Birkhahn RH et al. — Shock Index for Predicting Massive Transfusion (J Trauma, 2011)
- ›RCOG Green-top Guideline No. 52 — Prevention and Management of Postpartum Haemorrhage
- ›Mutschler M et al. — The Shock Index Revisited — A Fast Guide to Transfusion Requirement? (Critical Care, 2013)
- ›Cannon CM et al. — The GENESIS Project: Validation of a Modified Shock Index in Trauma (Ann Emerg Med, 2009)
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